FOXO4-DRI

FOXO4-D-Retro-Inverso

Anti-AgingNot ApprovedAnimalResearchSubQ

Popular for:Senolytic, eliminates senescent cells, anti-aging

0

Registered Trials

0

Trial Publications

17

PubMed References

Animal

Evidence Level

Overview

FOXO4-DRI is a D-retro-inverso peptide designed to selectively eliminate senescent cells (often called 'zombie cells') by disrupting the FOXO4-p53 interaction that keeps damaged cells alive. The short version: people usually care about it for senolytic, eliminates senescent cells, anti-aging, but the strength of the evidence depends heavily on indication and study type.

FOXO4-DRI is a D-retro-inverso peptide designed to selectively eliminate senescent cells (often called 'zombie cells') by disrupting the FOXO4-p53 interaction that keeps damaged cells alive. It was developed based on a landmark 2017 study published in Cell by Peter de Keizer's lab at Erasmus University Medical Center.

Senescent cells accumulate with age and secrete inflammatory factors (the SASP — senescence-associated secretory phenotype) that damage surrounding healthy tissue. FOXO4-DRI represents one of the most targeted senolytic approaches, designed to trigger apoptosis specifically in senescent cells while leaving healthy cells unaffected.

Research Snapshot

What the evidence says

Animal

FOXO4-DRI currently shows 0 registered trials from ClinicalTrials.gov, 0 PubMed trial publications (0 RCT-tagged), and 17 PubMed references matching the stored source query. Treat PubMed references as literature surface area, not a count of clinical trials.

Known vs uncertain

Known signals

  • 0 registered trials are tracked from ClinicalTrials.gov intervention records.
  • 0 PubMed clinical-trial publications are indexed.
  • 0 PubMed randomized controlled trial publications are indexed.
  • 17 PubMed references are tracked separately from trial counts and can include animal, in-vitro, review, mechanism, or clinical records.

Open questions

  • Evidence strength may vary by indication, route, formulation, and population.
  • Public anecdotes can highlight interest or concern but do not establish clinical efficacy.
  • Regulatory status and compounding access can change independently from the research literature.

Mechanism of Action

In senescent cells, FOXO4 binds to p53, sequestering it and preventing p53 from triggering apoptosis.

Key Research Benefits

Selectively targets and eliminates senescent cells
Based on peer-reviewed research (Cell, 2017)
Restored fur density and kidney function in aged mice
Targeted mechanism spares healthy cells
Represents cutting-edge senolytic research

Clinical Evidence Summary

Research Pipeline

Preclinical
Animal
Phase I
Phase II
Phase III
Approved

0

Registered Trials

0

Trial Publications

0

RCT Publications

17

PubMed References

ClinicalTrials.govPubMed ESearchExact-name queryChecked May 3, 2026

Registered trials are ClinicalTrials.gov intervention records. Trial publications are PubMed records tagged as clinical trials or randomized controlled trials. PubMed references are broader source-query matches and can include animal studies, in-vitro work, reviews, mechanism papers, and trial publications.

0

Registered trials

0

Trial publications

0

RCT publications

17

PubMed references

4

Reviews

0

Meta-analyses

Registered trials source

Jun 1, 2026

FOXO4-DRI

Uses the exact compound name as a ClinicalTrials.gov intervention query.

View source

Publication counts source

May 3, 2026

FOXO4-DRI

Uses the exact display name.

View source

Not FDA-approved. Pre-clinical research compound. Based on de Keizer et al., Cell 2017. No human clinical trials completed. One of the most expensive research peptides due to synthesis complexity.

Key PubMed References

Targeting the FOXO4-p53 axis by retro-inverso peptide senolytic agents: a pharmacological strategy to mitigate brain aging and cognitive decline.

Review

Alameen AAM, Al-Kuraishy HM, Fawzy MN, et al. · Naunyn-Schmiedeberg's archives of pharmacology · 2026

PMID: 42024235

The disordered p53 transactivation domain is the target of FOXO4 and the senolytic compound FOXO4-DRI.

In Vitro

Bourgeois B, Spreitzer E, Platero-Rochart D, et al. · Nature communications · 2025

PMID: 40593617

FOXO4-DRI induces keloid senescent fibroblast apoptosis by promoting nuclear exclusion of upregulated p53-serine 15 phosphorylation.

In Vitro

Kong YX, Li ZS, Liu YB, et al. · Communications biology · 2025

PMID: 39994346

FOXO4-DRI regulates endothelial cell senescence via the P53 signaling pathway.

In Vitro

Hu Z, Li F, Hu C, et al. · Frontiers in bioengineering and biotechnology · 2025

PMID: 41625068

Cellular Senescence Contributes to the Progression of Hyperoxic Bronchopulmonary Dysplasia.

Animal Study

Jing X, Jia S, Teng M, et al. · American journal of respiratory cell and molecular biology · 2024

PMID: 37874230

Anecdotes & Sentiment

Public discussion, not clinical evidence

This section summarizes what people are talking about in public sources. It can be useful for spotting questions, hype cycles, and recurring concerns, but it is separate from the evidence sections above.

No curated public-discussion themes are live for FOXO4-DRI yet.

Side Effects & Safety

- Limited human safety data - Potential for off-target effects not fully characterized - Research primarily in animal models - High cost due to complex D-retro-inverso synthesis

Limited human safety data
Potential for off-target effects not fully characterized
Research primarily in animal models
High cost due to complex D-retro-inverso synthesis

Known Interactions

No curated interaction entry is live for FOXO4-DRI yet.

Until the interaction table is fully populated, use the interaction checker and related peptides below to explore adjacent compounds and likely research pairings.

Comparison Pages

Comparison pages

All

No comparison page is linked yet.

Frequently Asked Questions

Research Disclaimer

This page is for research and educational purposes only. The information presented is based on published scientific literature and does not constitute medical advice, diagnosis, or treatment recommendations. Regulatory status can vary by compound, formulation, indication, and jurisdiction. Check official labeling, registry records, and qualified professional guidance before making any health-related decision. The studies referenced are linked to their original PubMed sources for verification.